Environmental Chemicals and Their Role in Obesity
NCT01778868 · Status: UNKNOWN · Type: OBSERVATIONAL · Enrollment: 203
Last updated 2013-01-29
Summary
Hormonal disruption is the inappropriate alteration of the hormonal system by chemical substances that are present in our environment. Some chemical substances are capable of replicating, enhancing or reducing the production, release, transport or action of natural hormones. Therefore, they are called hormonal or endocrine disrupters. Some 'classic' endocrine disrupters such as pesticides and dioxins are considered responsible for infertility, cancer and thyroid problems. Recently, a number of additional chemical substances were stipulated to have endocrine disrupting capabilities. The industrial production of these substances in large quantities has led to an accumulation in our environment and thus possible negative consequences on human health.
The aim of this study is to investigate the rol of these chemical substances in the occurence of overweight and obesity.
Conditions
Sponsors & Collaborators
-
Universiteit Antwerpen
collaborator OTHER -
University Hospital, Antwerp
lead OTHER
Principal Investigators
-
Philippe Jorens, MD, PhD · University Hospital, Antwerp
Eligibility
- Min Age
- 18 Years
- Sex
- ALL
- Healthy Volunteers
- Yes
Timeline & Regulatory
- Start
- 2009-08-31
- Primary Completion
- 2013-03-31
- Completion
- 2013-07-31
Countries
- Belgium
Study Locations
More Related Trials
-
Metabolism Associated With Abdominal Fat Mass Distribution
NCT01726647 ·Status: COMPLETED
-
Endothelial Dysfunction in Obese Children
NCT01461226 ·Status: COMPLETED ·Phase: NA
-
Low Grade Inflammation in Childhood Obesity : an Independent Risk Factor for Endothelial Dysfunction
NCT04181398 ·Status: COMPLETED ·Phase: NA
-
Weight-loss and Gut-brain Interactions
NCT01740050 ·Status: UNKNOWN ·Phase: NA
-
Course of Obesity and Extreme Obesity in Adolescents
NCT01662271 ·Status: COMPLETED
-
Endocrine Regulation of Lipolysis in Obesity and Diabetes
NCT02598544 ·Status: COMPLETED ·Phase: NA
-
Long Term Effects of Weight Loss on Post-prandial Gut Hormone Responses and Meal Induced Thermogenesis
NCT04170010 ·Status: UNKNOWN
-
Marin Protein Hydrolysate and Metabolic Syndrome
NCT03807752 ·Status: COMPLETED ·Phase: NA
-
Translational Study Using Human Abdominal Adipose Tissue Biopsies to Investigate the Role of Cannabinoid Receptor 1 (CB1) in Controlling Endocannabinoid and Adipokine Secretion
NCT04940962 ·Status: RECRUITING ·Phase: NA
-
The Impact of Obesity on Children and Adolescents.
NCT01231152 ·Status: UNKNOWN
-
Re-energize With Nutrition, Exercise and Weight Loss
NCT00712127 ·Status: COMPLETED ·Phase: NA
-
Modeling the Epidemiologic Transition Study
NCT02925156 ·Status: COMPLETED
-
Neuromolecular Risk Factors for Obesity (PROSPECT)
NCT03106688 ·Status: UNKNOWN ·Phase: NA
-
Pre- and Post-prandial Levels of Appetite Regulatory Hormones in Adults
NCT05815641 ·Status: COMPLETED ·Phase: NA
-
Healthy vs Unhealthy Obesity: Mehanistic Insights and Effects of Time-Restricted Eating
NCT05136313 ·Status: COMPLETED ·Phase: NA
-
A Translational Understanding of Obesity-Related Phenotypes Using Brain Imaging and Manipulation
NCT06353464 ·Status: RECRUITING ·Phase: NA
-
Adiposity and Endothelin Receptor Function
NCT03583866 ·Status: ACTIVE_NOT_RECRUITING ·Phase: EARLY_PHASE1
-
EAT-Lancet Diet, Chronotype, and Well-Being in Obesity
NCT07168967 ·Status: COMPLETED
-
Regulation of Energy Balance and Metabolism - Mechanisms Behind and Beyond Obesity and Weight Loss
NCT03550339 ·Status: COMPLETED ·Phase: NA
-
The Role of Atrial Natriuretic Peptide in the Lipolytic Process: Effect of Obesity and Exercise
NCT02418728 ·Status: COMPLETED
-
Anthropometric Assessment of Abdominal Obesity and Health Risk in Children and Adolescents
NCT01595100 ·Status: COMPLETED
-
Metabolic Phenotyping for Personalized Obesity Therapy
NCT06874270 ·Status: RECRUITING
-
Physical and Behavioral Traits of Overweight and Obese Adults
NCT00428987 ·Status: RECRUITING
-
Identification and Characterization of Youth With Extreme Obesity
NCT01625325 ·Status: COMPLETED
-
Study of the Mechanisms of Metabolic Adaptations to Overfeeding
NCT02225457 ·Status: UNKNOWN ·Phase: NA