Characterizing PAI-1 Modulation on Monocyte Adhesion
NCT00512031 · Status: TERMINATED · Type: OBSERVATIONAL · Enrollment: 50
Last updated 2011-07-18
Summary
To determine how altering the expression of a gene known as PAI-1 may affect the adhesive capacity of cells that play a critical role in the developement of human atherosclerosis.
Conditions
Sponsors & Collaborators
-
Vanderbilt University
lead OTHER
Principal Investigators
-
Mohan Sathyamoorthy, MD · Vanderbilt University
Eligibility
- Min Age
- 18 Years
- Max Age
- 50 Years
- Sex
- ALL
- Healthy Volunteers
- Yes
Timeline & Regulatory
- Start
- 2007-01-31
- Primary Completion
- 2011-07-31
- Completion
- 2011-07-31
Countries
- United States
Study Locations
More Related Trials
-
Molecular Determinants of Coronaruy Artery Disease
NCT00590291 ·Status: TERMINATED
-
Risk and Resilience in Pulmonary Arterial Hypertension and Genetically Susceptible Individuals
NCT05584722 ·Status: RECRUITING
-
Gene-by-Smoking Interactions and Risk of Atherosclerosis - Ancillary to ARIC
NCT00064545 ·Status: COMPLETED
-
Understanding the Genetic Basis of Atherosclerosis and Peripheral Arterial Disease
NCT00380185 ·Status: COMPLETED
-
Molecular Investigation of Genetic Factors in CArDiovascular Diseases Using an BIOresource of Healthy Volunteers
NCT03038750 ·Status: TERMINATED
-
Understanding the Genetic and Hereditary Basis of Atherosclerosis
NCT00344292 ·Status: COMPLETED
-
Apolipoprotein A-I Gene Polymorphism and Atherosclerosis
NCT00005183 ·Status: COMPLETED
-
Protein and Phospholipid Analysis of HDL in Patients With Very High Serum Levels of HDL-C
NCT00525083 ·Status: TERMINATED
-
Epigenetic Reprogramming of Monocytes in Patients With Coronary Atherosclerosis
NCT02393768 ·Status: COMPLETED
-
Atherosclerosis, Plaque and CVD in Communities
NCT00091754 ·Status: COMPLETED
-
Identification of Genes and Pathogenesis Involved in Familial Thoracic Aortic Aneurysm
NCT02256163 ·Status: COMPLETED
-
Peking University & University of Michigan Study of MI and Atherosclerosis
NCT04950699 ·Status: COMPLETED
-
Longitudinal Interaction Between APOA5 -1131T>C and Overweight Accelerates Arterial Stiffness
NCT03367598 ·Status: COMPLETED
-
The Effects of Renal Function and Atrial Fibrillation on Lipoproteins and Clot Structure/Function
NCT04043026 ·Status: TERMINATED
-
Association Study to Evaluate TFPI Gene in CAD in Han Chinese
NCT02961127 ·Status: UNKNOWN
-
Association Between Levels of D-Dimer, Fibrinogen and PAI-1 in Elderly Patients With Infection and Occurrence of Cardio- and Cerebro-vascular Disease After Discharge
NCT00512603 ·Status: COMPLETED
-
Genetic- and Biological-Markers of Predisposition to Atherosclerosis: Leipzig-Heart Study
NCT00497887 ·Status: UNKNOWN
-
Advanced Lipoproptein Profiling and Cardiovascular Risk Stratification in Familial Hypercholesterolemia
NCT05066932 ·Status: UNKNOWN
-
Mechanisms of Disability in Peripheral Arterial Disease
NCT00046592 ·Status: COMPLETED
-
Molecular & Clinical Evaluation of Low HDL Syndromes
NCT00006295 ·Status: COMPLETED
-
Identification of Genes Predisposing to Atherosclerosis
NCT00059098 ·Status: COMPLETED
-
Modeling DNA Diversity in Reverse Cholesterol Transport
NCT00064493 ·Status: COMPLETED
-
Assessment of Lipoprotein(a) and Endogenous Fibrinolysis in Atherosclerotic Cardiovascular Disease/Aortic Valve Disease
NCT06126367 ·Status: RECRUITING
-
Involvement of Monocytic B1 and B2 Receptors in Inflammation and Chronic Vascular Disease in Patients With Hereditary Bradykinetic Angioedema
NCT04032002 ·Status: COMPLETED ·Phase: NA
-
Inflammatory Genomics in Human Carotid Artery Disease
NCT00070668 ·Status: COMPLETED