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Selenoprotein P and Non-alcoholic Fatty Liver Disease

NCT01257685 · Status: COMPLETED · Type: OBSERVATIONAL · Enrollment: 120

Last updated 2020-09-03

No results posted yet for this study

Summary

The pathogenesis of nonalcoholic fatty liver disease has not been fully elucidated. The most widely supported theory implicates insulin resistance as the key mechanism leading to hepatic steatosis, and perhaps also to steatohepatitis.

Selenoprotein P(SeP) is a secretory protein primarily produced by the liver. Previous studies demonstrated that SeP, a liver-derived secretory protein, causes insulin resistance.

Therefore, the purpose of this study is to determine the different Sep levels between healthy normal group and NAFLD group.

Conditions

Sponsors & Collaborators

  • Korea University

    lead OTHER

Principal Investigators

  • Kyung Mook Choi, MD.PhD · Korea University

Eligibility

Min Age
20 Years
Max Age
80 Years
Sex
ALL
Healthy Volunteers
Yes

Timeline & Regulatory

Start
2007-09-30
Primary Completion
2008-08-31
Completion
2008-08-31

Countries

  • South Korea

Study Locations

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Entities

Diseases

Read the full study record

This page highlights key information. For complete eligibility criteria, study locations, investigator contacts, and the full protocol, visit the original record on ClinicalTrials.gov.

View NCT01257685 on ClinicalTrials.gov