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Oxidation-reduction Homeostasis in Cognitive Impairment Patients After Stroke

NCT03519828 · Status: COMPLETED · Type: OBSERVATIONAL · Enrollment: 100

Last updated 2019-07-31

No results posted yet for this study

Summary

Cognitive impairment after ischemic stroke can affect not only the social adaptation ability, but also affect the comprehensive rehabilitation of patients. The damage of cognitive impairment after ischemic stroke is not lower than the body function defect after stroke. Many studies have shown that oxidative stress is one of the pathophysiological mechanisms of ischemic cerebrovascular disease. Many studies have reported that the oxidative-reduction of cells plays an important role in the life activities of organisms, affecting the health, aging and death of the organism. In the recent years, some scholars have suggested that post-stroke cognitive impairment may be related to oxidative-reduction homeostasis of the body, but the relevant evidence is lacking and needs to be further explored. Therefore, the purpose of this study is to explore the effect of oxidation-reduction homeostasis on cognitive impairment in patients with ischemic stroke, and provide a theoretical basis for the prevention and treatment of cognitive impairment after ischemic stroke.

Conditions

  • Oxidative-reduction Homeostasis
  • Post-stroke Cognitive Impairment

Interventions

OTHER

No intervention has been conducted

It is an observational study, and no intervention has been conducted

Sponsors & Collaborators

  • Hui-Sheng Chen

    lead OTHER

Eligibility

Min Age
40 Years
Max Age
80 Years
Sex
ALL
Healthy Volunteers
No

Timeline & Regulatory

Start
2018-03-01
Primary Completion
2018-12-30
Completion
2019-01-01

Countries

  • China

Study Locations

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Read the full study record

This page highlights key information. For complete eligibility criteria, study locations, investigator contacts, and the full protocol, visit the original record on ClinicalTrials.gov.

View NCT03519828 on ClinicalTrials.gov