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The Mechanism of TCF7L2 and SLC30A8 on Antipsychotic-induced Metabolic Syndrome

NCT02093858 · Status: UNKNOWN · Type: OBSERVATIONAL · Enrollment: 212

Last updated 2014-03-21

No results posted yet for this study

Summary

Almost all of antipsychotics can induce metabolic syndrome,Genetic factors play a key role in the development of metabolic syndrome,TCF7L2 and SLC30A8 are strongestly correlated with metabolic syndrome.Moreover,Antipsychotics have an effect on the expression of TCF7L2 and SLC30A8 genes.It indicates the variations of TCF7L2 and SLC30A8 play an important part in the development of antipsychotics-induced metabolic syndrome.

Conditions

Sponsors & Collaborators

  • Central South University

    lead OTHER

Principal Investigators

  • Wu R Rong, PhD · Central South University

Eligibility

Min Age
18 Years
Max Age
45 Years
Sex
ALL
Healthy Volunteers
No

Timeline & Regulatory

Start
2014-04-30
Primary Completion
2017-12-31
Completion
2017-12-31

Countries

  • China

Study Locations

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Entities

Diseases

Read the full study record

This page highlights key information. For complete eligibility criteria, study locations, investigator contacts, and the full protocol, visit the original record on ClinicalTrials.gov.

View NCT02093858 on ClinicalTrials.gov