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The Role of Angiotensinogen Gene Polymorphism in the Pathogenesis of Non-familial Sick Sinus Syndrome

NCT01310920 · Status: UNKNOWN · Type: OBSERVATIONAL · Enrollment: 400

Last updated 2011-03-09

No results posted yet for this study

Summary

Evidence has been documented for the close relationship between the arrhythmia pathogenesis and the gene expression in renin-angiotensin system. However, it remains unclear for involvement of RAS in the pathogenesis of non-familial sick sinus syndrome. The researchers thus investigated the possible relationship between non-familial sick sinus syndrome and the polymorphism and haplotype of the AGT promoter.

Conditions

  • Sick Sinus Syndrome

Interventions

OTHER

observation

genotyping

Sponsors & Collaborators

  • China Medical University Hospital

    lead OTHER

Principal Investigators

  • Jan-Yow Chen, M.D. · China Medical University Hospital

Eligibility

Sex
ALL
Healthy Volunteers
Yes

Timeline & Regulatory

Start
2011-02-28

Countries

  • Taiwan

Study Locations

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Read the full study record

This page highlights key information. For complete eligibility criteria, study locations, investigator contacts, and the full protocol, visit the original record on ClinicalTrials.gov.

View NCT01310920 on ClinicalTrials.gov